At this time, there is no known cure for postaxial polydactyly type B. However, there are several surgical options available to remove the extra digits. Medications are not typically used to treat this condition.

1. Family history of postaxial polydactyly type B
2. Maternal diabetes
3. Maternal use of certain medications during pregnancy
4. Maternal alcohol consumption during pregnancy
5. Maternal smoking during pregnancy
6. Maternal exposure to certain environmental toxins during pregnancy
7. Genetic mutations or chromosomal abnormalities

1. Surgery: Surgery is the most common treatment for postaxial polydactyly type B. The extra digit is removed and the remaining digits are realigned.

2. Splinting: Splinting may be used to help realign the digits and prevent the extra digit from growing back.

3. Physical Therapy: Physical therapy may be used to help strengthen the muscles and tendons in the affected area.

4. Orthotics: Orthotics may be used to help support the affected area and prevent further deformity.

5. Medication: Medication may be prescribed to help reduce pain and inflammation.

Postaxial polydactyly type B is caused by a genetic mutation in the HOXD13 gene. This gene is responsible for the development of the limbs and digits. Mutations in this gene can cause the extra digits to form. It is also believed that environmental factors, such as exposure to certain chemicals, may play a role in the development of this condition.

Symptoms of Postaxial polydactyly type B include the presence of extra fingers or toes on the outer side of the hand or foot, respectively. The extra digits may be fully formed or may be webbed or fused together. Other symptoms may include a wide space between the big toe and the second toe, and the extra digits may be smaller than the other digits.

Postaxial polydactyly type B is a congenital condition in which a person is born with an extra digit on the little finger side of the hand or foot. It is caused by a mutation in the HOXD13 gene. This condition is usually inherited in an autosomal dominant pattern, meaning that only one copy of the mutated gene is needed for a person to be affected.