At this time, there is no cure for Primary CD59 deficiency. However, medications such as intravenous immunoglobulin (IVIG) and eculizumab (Soliris) may be used to reduce the risk of serious complications associated with the condition.

The primary risk factor for Primary CD59 deficiency is a genetic mutation in the CD59 gene. This mutation is inherited in an autosomal recessive pattern, meaning that both parents must pass on the mutated gene for a child to be affected. Other risk factors include a family history of the condition, being of Ashkenazi Jewish descent, and having a weakened immune system.

The primary treatment for Primary CD59 deficiency is a lifelong regimen of intravenous immunoglobulin (IVIG) therapy. This therapy helps to replace the missing CD59 protein and reduce the risk of recurrent infections. Other treatments may include antibiotics, antiviral medications, and antifungal medications to treat any infections that may occur. In some cases, a bone marrow transplant may be recommended to replace the defective CD59 gene.

Primary CD59 deficiency is caused by mutations in the CD59 gene, which is responsible for producing the CD59 protein. This protein is important for protecting red blood cells from destruction by the body's own immune system. Mutations in the CD59 gene can lead to a decrease in the amount of CD59 protein produced, resulting in an increased risk of red blood cell destruction.

The symptoms of Primary CD59 deficiency vary from person to person, but may include recurrent infections, anemia, thrombocytopenia (low platelet count), and an increased risk of developing autoimmune diseases such as lupus. Other symptoms may include fatigue, joint pain, skin rashes, and kidney problems.

Primary CD59 deficiency is a rare genetic disorder caused by a mutation in the CD59 gene. This gene is responsible for producing a protein called CD59, which is important for protecting cells from damage caused by the body's own immune system. People with this disorder have a deficiency of CD59, which can lead to an increased risk of autoimmune diseases, such as lupus, and an increased risk of infections.