At this time, there is no known cure for PEHO-like syndrome. However, there are medications that can help manage the symptoms of the disorder. These include anticonvulsants, muscle relaxants, and medications to help with sleep disturbances. Additionally, physical and occupational therapy can help improve motor skills and coordination.

1. Genetic mutations in the SLC25A4 gene
2. Family history of PEHO-like syndrome
3. Low birth weight
4. Premature birth
5. Exposure to certain environmental toxins
6. Exposure to certain medications during pregnancy
7. Maternal infections during pregnancy
8. Maternal diabetes during pregnancy

The treatments for PEHO-like syndrome vary depending on the individual and the severity of the condition. Generally, treatment focuses on managing the symptoms and preventing further complications. This may include physical therapy, occupational therapy, speech therapy, medications, dietary modifications, and lifestyle changes. In some cases, surgery may be necessary to correct physical deformities or to improve mobility.

PEHO-like syndrome is a rare genetic disorder caused by mutations in the POLR3A gene. This gene is responsible for the production of a protein called polymerase III, which is involved in the production of proteins in the body. Mutations in this gene can lead to a variety of symptoms, including seizures, intellectual disability, and movement disorders. Other causes of PEHO-like syndrome include mutations in the POLR3B gene, which is involved in the production of a different type of polymerase, and mutations in the POLR3G gene, which is involved in the production of a third type of polymerase.

The symptoms of PEHO-like syndrome vary from person to person, but may include:

-Developmental delay
-Seizures
-Intellectual disability
-Movement disorders
-Growth retardation
-Feeding difficulties
-Hearing loss
-Vision problems
-Skin abnormalities
-Behavioral problems
-Sleep disturbances
-Gastrointestinal issues
-Cardiac abnormalities

PEHO-like syndrome is a rare neurological disorder characterized by seizures, developmental delay, and hypotonia (low muscle tone). It is caused by mutations in the EFHC1 gene, which is involved in the regulation of neuronal development and function. Symptoms may include delayed motor development, intellectual disability, and seizures. Treatment typically involves anticonvulsant medications and physical and occupational therapy.